Additionally, both types of studies depend on particular assumptions, such as the equal environments assumption in the case of twin studies, and the lack of pre-adoptive effects in the case of adoption studies. Since the definition of "nature" in this context is tied to "heritability", the definition of "nurture" has necessarily become very wide, including any type of causality that is not heritable. The term has thus moved away from its original connotation of "cultural influences" to include all effects of the environment, including; indeed, a substantial source of environmental input to human nature may arise from stochastic variations in prenatal development and is thus in no sense of the term "cultural".
Heritability refers to the origins of differences between people. Individual development, even of highly heritable traits, such as eye color, depends on a range of environmental factors, from the other genes in the organism, to physical variables such as temperature, oxygen levels etc. The variability of trait can be meaningfully spoken of as being due in certain proportions to genetic differences "nature" , or environments "nurture".
For highly penetrant Mendelian genetic disorders such as Huntington's disease virtually all the incidence of the disease is due to genetic differences. Huntington's animal models live much longer or shorter lives depending on how they are cared for [ citation needed ].
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At the other extreme, traits such as native language are environmentally determined: linguists have found that any child if capable of learning a language at all can learn any human language with equal facility. At a molecular level, genes interact with signals from other genes and from the environment.
While there are many thousands of single-gene-locus traits, so-called complex traits are due to the additive effects of many often hundreds of small gene effects. A good example of this is height, where variance appears to be spread across many hundreds of loci. Extreme genetic or environmental conditions can predominate in rare circumstances—if a child is born mute due to a genetic mutation, it will not learn to speak any language regardless of the environment; similarly, someone who is practically certain to eventually develop Huntington's disease according to their genotype may die in an unrelated accident an environmental event long before the disease will manifest itself.
Steven Pinker likewise described several examples:  . When traits are determined by a complex interaction of genotype and environment it is possible to measure the heritability of a trait within a population.
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However, many non-scientists who encounter a report of a trait having a certain percentage heritability imagine non-interactional, additive contributions of genes and environment to the trait. As an analogy, some laypeople may think of the degree of a trait being made up of two "buckets," genes and environment, each able to hold a certain capacity of the trait. But even for intermediate heritabilities, a trait is always shaped by both genetic dispositions and the environments in which people develop, merely with greater and lesser plasticities associated with these heritability measures.
Heritability measures always refer to the degree of variation between individuals in a population. That is, as these statistics cannot be applied at the level of the individual, it would be incorrect to say that while the heritability index of personality is about 0. To help to understand this, imagine that all humans were genetic clones. The heritability index for all traits would be zero all variability between clonal individuals must be due to environmental factors.
And, contrary to erroneous interpretations of the heritability index, as societies become more egalitarian everyone has more similar experiences the heritability index goes up as environments become more similar, variability between individuals is due more to genetic factors. One should also take into account the fact that the variables of heritability and environmentality are not precise and vary within a chosen population and across cultures.
Nature and Nurture
It would be more accurate to state that the degree of heritability and environmentality is measured in its reference to a particular phenotype in a chosen group of a population in a given period of time. The accuracy of the calculations is further hindered by the number of coefficients taken into consideration, age being one such variable. The display of the influence of heritability and environmentality differs drastically across age groups: the older the studied age is, the more noticeable the heritability factor becomes, the younger the test subjects are, the more likely it is to show signs of strong influence of the environmental factors.
Some have pointed out that environmental inputs affect the expression of genes  see the article on epigenetics. This is one explanation of how environment can influence the extent to which a genetic disposition will actually manifest. A classic example of gene—environment interaction is the ability of a diet low in the amino acid phenylalanine to partially suppress the genetic disease phenylketonuria. Yet another complication to the nature—nurture debate is the existence of gene—environment correlations.
These correlations indicate that individuals with certain genotypes are more likely to find themselves in certain environments. Thus, it appears that genes can shape the selection or creation of environments. Even using experiments like those described above, it can be very difficult to determine convincingly the relative contribution of genes and environment. A study conducted by T.
Bouchard, Jr. The results shown have been important evidence against the importance of environment when determining, happiness, for example. In the Minnesota study of twins reared apart, it was actually found that there was higher correlation for monozygotic twins reared apart 0.
Nature versus nurture - Wikipedia
Also, highlighting the importance of genes, these correlations found much higher correlation among monozygotic than dizygotic twins that had a correlation of 0. The social pre-wiring hypothesis refers to the ontogeny of social interaction. Also informally referred to as, "wired to be social. Research in the theory concludes that newborns are born into the world with a unique genetic wiring to be social. Circumstantial evidence supporting the social pre-wiring hypothesis can be revealed when examining newborns' behavior.
Newborns, not even hours after birth, have been found to display a preparedness for social interaction. This preparedness is expressed in ways such as their imitation of facial gestures. This observed behavior cannot be contributed to any current form of socialization or social construction.
Rather, newborns most likely inherit to some extent social behavior and identity through genetics. Principal evidence of this theory is uncovered by examining twin pregnancies. The main argument is, if there are social behaviors that are inherited and developed before birth, then one should expect twin foetuses to engage in some form of social interaction before they are born. Thus, ten foetuses were analyzed over a period of time using ultrasound techniques. Using kinematic analysis, the results of the experiment were that the twin foetuses would interact with each other for longer periods and more often as the pregnancies went on.
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Researchers were able to conclude that the performance of movements between the co-twins were not accidental but specifically aimed. The social pre-wiring hypothesis was proved correct, "The central advance of this study is the demonstration that ' social actions ' are already performed in the second trimester of gestation. Starting from the 14th week of gestation twin foetuses plan and execute movements specifically aimed at the co-twin. These findings force us to predate the emergence of social behavior : when the context enables it, as in the case of twin foetuses, other-directed actions are not only possible but predominant over self-directed actions.
Traits may be considered to be adaptations such as the umbilical cord , byproducts of adaptations the belly button or due to random variation convex or concave belly button shape. For example, the rewarding sweet taste of sugar and the pain of bodily injury are obligate psychological adaptations—typical environmental variability during development does not much affect their operation.
The attachment style of adults, for example, a "secure attachment style," the propensity to develop close, trusting bonds with others is proposed to be conditional on whether an individual's early childhood caregivers could be trusted to provide reliable assistance and attention.
An example of a facultative physiological adaptation is tanning of skin on exposure to sunlight to prevent skin damage. Facultative social adaptation have also been proposed. For example, whether a society is warlike or peaceful has been proposed to be conditional on how much collective threat that society is experiencing .
Developmental genetic analysis examines the effects of genes over the course of a human lifespan. Subsequent developmental genetic analyses found that variance attributable to additive environmental effects is less apparent in older individuals,    with estimated heritability of IQ increasing in adulthood. Multivariate genetic analysis examines the genetic contribution to several traits that vary together.
For example, multivariate genetic analysis has demonstrated that the genetic determinants of all specific cognitive abilities e. Similarly, multivariate genetic analysis has found that genes that affect scholastic achievement completely overlap with the genes that affect cognitive ability.
Extremes analysis examines the link between normal and pathological traits. For example, it is hypothesized that a given behavioral disorder may represent an extreme of a continuous distribution of a normal behavior and hence an extreme of a continuous distribution of genetic and environmental variation. Depression, phobias, and reading disabilities have been examined in this context. For a few highly heritable traits, studies have identified loci associated with variance in that trait, for instance in some individuals with schizophrenia.
Through studies of identical twins separated at birth, one-third of their creative thinking abilities come from genetics and two-thirds come from learning. Evidence from behavioral genetic research suggests that family environmental factors may have an effect upon childhood IQ , accounting for up to a quarter of the variance.
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The American Psychological Association's report " Intelligence: Knowns and Unknowns " states that there is no doubt that normal child development requires a certain minimum level of responsible care. Here, environment is playing a role in what is believed to be fully genetic intelligence but it was found that severely deprived, neglectful, or abusive environments have highly negative effects on many aspects of children's intellect development. Beyond that minimum, however, the role of family experience is in serious dispute. On the other hand, by late adolescence this correlation disappears, such that adoptive siblings no longer have similar IQ scores.
Moreover, adoption studies indicate that, by adulthood, adoptive siblings are no more similar in IQ than strangers IQ correlation near zero , while full siblings show an IQ correlation of 0.